RottenRoots
Active Member
Lately noticed thrips on my clones I took from the big girls before they went to 12/12. The thrips are hiding on the underside of the lower fan leaves, which is why I never really noticed them. When I found em on my clones I went to check the tent and realized I had the unwanted silver marks from those damn bastards. At first I was going to grab some hotshot pest strips but after researching, don't really won't those around me or the plants. Most people won't spray their flowers during flower and I'm skeptical about it as well. (I am on my 5th week of 12/12, 3rd week of flowering. )
Here's what I found.
[SUP]
[/SUP]Spinosad is highly active, by both contact and ingestion, to numerous pests.[SUP][5][/SUP] Spinosad’s overall protective effect varies with pest species and life stage. Spinosad affects certain insect pests only in the adult stage, but can affect other pests at more than one life stage. The pests that are subject to very high rates of mortality as larvae, but not as adults, may gradually be controlled through sustained larval mortality.[SUP][5][/SUP] The mode of action of spinosoid insecticides is via a neural mechanism.[SUP][6][/SUP] The spinosyns and spinosoids have a novel mode of action, primarily targeting binding sites on nicotinic acetylcholine receptors (nAChRs) of the insect nervous system that are distinct from those at which other insecticides have their activity. Spinosoid binding leads to disruption of acetylcholine neurotransmission.[SUP][2][/SUP] Spinosad also has secondary effects as a Ɣ-amino-butyric acid (GABA) neurotransmitter agonist.[SUP][2][/SUP] Spinosad kills insects via hyperexcitation of the insect nervous system.[SUP][2][/SUP] Spinosad so far has proven non cross-resistant to any other known insecticide.[SUP][7][/SUP]
Spinosad has been used around the world for the control of a variety of insect pests, including Lepidoptera, Diptera, Thysanoptera, Coleoptera, Orthoptera and Hymenoptera, and many others.[SUP][8][/SUP] Spinosad was first registered as a pesticide in the United States for use on crops in 1997.[SUP][8][/SUP] Its labeled use rate is set at 1 ppm (1 mg a.i./kg of grain) and its Maximum Residue Limit (MRL) or tolerance is set at 1.5 ppm. Spinosad’s widespread commercial launch was deferred, awaiting final MRL or tolerance approvals in a few remaining grain-importing countries. Spinosad is considered a natural product, and thus is approved for use in organic agriculture by numerous nations.[SUP][5][/SUP] Two other uses for Spinosad are for pets and humans. Spinosad has recently been used in oral preparations to treat C. felis, the cat flea, in canines and felines; the optimal dose set for canines is reported to be 30 mg/kg.[SUP][2][/SUP] Brand names include Comfortis and Trifexis® (which also includes milbemycin oxime) (both brands treat adult fleas on pets; the latter also prevents heartworm disease), and Natroba (for human head lice)[SUP][9][/SUP][SUP][10][/SUP][SUP][11]
[/SUP]Spinosyn A does not appear to interact directly with known insecticidal-relevant target sites, but rather acts via a novel mechanism.[SUP][6][/SUP] Spinosyn A resembles a GABA antagonist and is comparable to the effect of avermectin on insect neurons.[SUP][4][/SUP] Spinosyn A is highly active against neonate larvae of the tobacco budworm, Heliothis virescens, and is slightly more biologically active than Spinosyn D. In general spinosyns possessing a methyl group at C6 (Spinosyn D-related analogs) tend to be more active and less affected by changes in the rest of the molecule.[SUP][7][/SUP] Spinosyn A is slow to penetrate to the internal fluids of larvae; it is also poorly metabolized once it enters the insect.[SUP][7][/SUP] The apparent lack of Spinosyn A metabolism may contribute to its high level of activity, and may compensate for the slow rate of penetration.[SUP][7][/SUP]
Spinosad has high efficacy, a broad insect pest spectrum, low mammalian toxicity, and a good environmental profile. This is a unique feature of an insecticide, compared to others that are currently used for the protection of grain products.[SUP][5][/SUP] Spinosad is considered a natural product and approved for use in organic agriculture by numerous national and international certifications.[SUP][8][/SUP] Spinosad residues are highly stable on grains stored in bins, with protection ranging from 6 months to 2 years.[SUP][5][/SUP] Ecotoxicology parameters for Spinosad are for Rat (Rattus norvegicus Bergenhout, 1769)-acute oral: LD[SUB]50[/SUB]>5000 mg/kg (non-toxic), Rat (Rattus norvegicus Bergenhout, 1769)-acute dermal: LD[SUB]50[/SUB]>2000 mg/kg (non-toxic), Quail (Calliperla californica Shaw, 179
-oral toxicity: LD[SUB]50[/SUB]>2000 mg/kg (non-toxic), Duck (Anas platyrhynchos domestica Linnaeus, 175-dietary toxicity: LC[SUB]50[/SUB]>5000 mg/kg (non-toxic), Rainbow Trout (Oncorhynchus mykiss Walbaum, 1792): LC[SUB]50-96h[/SUB]=30,0 mg/l (slightly toxic), Honeybee (Apis mellifera Linnaeus, 175: LD[SUB]50[/SUB]=0,0025 mg/bee (highly toxic, if directly sprayed on bees, but dried residues show little toxicity).[SUP][12][/SUP]
However, most tests ignore the cytotoxic(toxicity within the cell) aspects. One study,"Cytogenetic and Hormonal Alteration in Rats Exposed to Recommended “Safe Doses” of Spinosad and Malathion Insecticides", by S.A. MANSOUR1, A.H. MOSSA AND T.M. HEIKAL, found significant markers for long term mutagenic effects in mammals.[SUP][13][/SUP]
Regardless of the above findings, chronic exposure studies failed to induce tumor formation in rats and mice. Mice given up to 51mg/kg/day for 18 months resulted in no tumor formation. http://toxsci.oxfordjournals.org/content/65/2/276.full . Similarly, rats given 25mg/kg/day for 24 months did not experience tumors. http://toxsci.oxfordjournals.org/content/65/2/288.full
Doesn't sound like it will harm my plant's or me from smoking non-toxic bacteria compounds. The MRL is 1.5 ppm which could probably just be washed off with water. Besides it's non-toxic to almost all mammals. Cherry farmers use them on their cherries, would their be any difference from eting the residue or smoking it? I'm thinking spraying in flower with an organic compound can't possibly be that bad, compared to throwing a no pest strip in there, or using other types of toxic chemicals. Anyway would like to hear some opinions.
Here's what I found.
[SUP]
[/SUP]Spinosad is highly active, by both contact and ingestion, to numerous pests.[SUP][5][/SUP] Spinosad’s overall protective effect varies with pest species and life stage. Spinosad affects certain insect pests only in the adult stage, but can affect other pests at more than one life stage. The pests that are subject to very high rates of mortality as larvae, but not as adults, may gradually be controlled through sustained larval mortality.[SUP][5][/SUP] The mode of action of spinosoid insecticides is via a neural mechanism.[SUP][6][/SUP] The spinosyns and spinosoids have a novel mode of action, primarily targeting binding sites on nicotinic acetylcholine receptors (nAChRs) of the insect nervous system that are distinct from those at which other insecticides have their activity. Spinosoid binding leads to disruption of acetylcholine neurotransmission.[SUP][2][/SUP] Spinosad also has secondary effects as a Ɣ-amino-butyric acid (GABA) neurotransmitter agonist.[SUP][2][/SUP] Spinosad kills insects via hyperexcitation of the insect nervous system.[SUP][2][/SUP] Spinosad so far has proven non cross-resistant to any other known insecticide.[SUP][7][/SUP]
Spinosad has been used around the world for the control of a variety of insect pests, including Lepidoptera, Diptera, Thysanoptera, Coleoptera, Orthoptera and Hymenoptera, and many others.[SUP][8][/SUP] Spinosad was first registered as a pesticide in the United States for use on crops in 1997.[SUP][8][/SUP] Its labeled use rate is set at 1 ppm (1 mg a.i./kg of grain) and its Maximum Residue Limit (MRL) or tolerance is set at 1.5 ppm. Spinosad’s widespread commercial launch was deferred, awaiting final MRL or tolerance approvals in a few remaining grain-importing countries. Spinosad is considered a natural product, and thus is approved for use in organic agriculture by numerous nations.[SUP][5][/SUP] Two other uses for Spinosad are for pets and humans. Spinosad has recently been used in oral preparations to treat C. felis, the cat flea, in canines and felines; the optimal dose set for canines is reported to be 30 mg/kg.[SUP][2][/SUP] Brand names include Comfortis and Trifexis® (which also includes milbemycin oxime) (both brands treat adult fleas on pets; the latter also prevents heartworm disease), and Natroba (for human head lice)[SUP][9][/SUP][SUP][10][/SUP][SUP][11]
[/SUP]Spinosyn A does not appear to interact directly with known insecticidal-relevant target sites, but rather acts via a novel mechanism.[SUP][6][/SUP] Spinosyn A resembles a GABA antagonist and is comparable to the effect of avermectin on insect neurons.[SUP][4][/SUP] Spinosyn A is highly active against neonate larvae of the tobacco budworm, Heliothis virescens, and is slightly more biologically active than Spinosyn D. In general spinosyns possessing a methyl group at C6 (Spinosyn D-related analogs) tend to be more active and less affected by changes in the rest of the molecule.[SUP][7][/SUP] Spinosyn A is slow to penetrate to the internal fluids of larvae; it is also poorly metabolized once it enters the insect.[SUP][7][/SUP] The apparent lack of Spinosyn A metabolism may contribute to its high level of activity, and may compensate for the slow rate of penetration.[SUP][7][/SUP]
Spinosad has high efficacy, a broad insect pest spectrum, low mammalian toxicity, and a good environmental profile. This is a unique feature of an insecticide, compared to others that are currently used for the protection of grain products.[SUP][5][/SUP] Spinosad is considered a natural product and approved for use in organic agriculture by numerous national and international certifications.[SUP][8][/SUP] Spinosad residues are highly stable on grains stored in bins, with protection ranging from 6 months to 2 years.[SUP][5][/SUP] Ecotoxicology parameters for Spinosad are for Rat (Rattus norvegicus Bergenhout, 1769)-acute oral: LD[SUB]50[/SUB]>5000 mg/kg (non-toxic), Rat (Rattus norvegicus Bergenhout, 1769)-acute dermal: LD[SUB]50[/SUB]>2000 mg/kg (non-toxic), Quail (Calliperla californica Shaw, 179
-oral toxicity: LD[SUB]50[/SUB]>2000 mg/kg (non-toxic), Duck (Anas platyrhynchos domestica Linnaeus, 175-dietary toxicity: LC[SUB]50[/SUB]>5000 mg/kg (non-toxic), Rainbow Trout (Oncorhynchus mykiss Walbaum, 1792): LC[SUB]50-96h[/SUB]=30,0 mg/l (slightly toxic), Honeybee (Apis mellifera Linnaeus, 175: LD[SUB]50[/SUB]=0,0025 mg/bee (highly toxic, if directly sprayed on bees, but dried residues show little toxicity).[SUP][12][/SUP]However, most tests ignore the cytotoxic(toxicity within the cell) aspects. One study,"Cytogenetic and Hormonal Alteration in Rats Exposed to Recommended “Safe Doses” of Spinosad and Malathion Insecticides", by S.A. MANSOUR1, A.H. MOSSA AND T.M. HEIKAL, found significant markers for long term mutagenic effects in mammals.[SUP][13][/SUP]
Regardless of the above findings, chronic exposure studies failed to induce tumor formation in rats and mice. Mice given up to 51mg/kg/day for 18 months resulted in no tumor formation. http://toxsci.oxfordjournals.org/content/65/2/276.full . Similarly, rats given 25mg/kg/day for 24 months did not experience tumors. http://toxsci.oxfordjournals.org/content/65/2/288.full
Doesn't sound like it will harm my plant's or me from smoking non-toxic bacteria compounds. The MRL is 1.5 ppm which could probably just be washed off with water. Besides it's non-toxic to almost all mammals. Cherry farmers use them on their cherries, would their be any difference from eting the residue or smoking it? I'm thinking spraying in flower with an organic compound can't possibly be that bad, compared to throwing a no pest strip in there, or using other types of toxic chemicals. Anyway would like to hear some opinions.
